Monday, September 3, 2012

Bandra Fish Market

The Bandra Fish market is the domain of the Koli fisherwomen, who come here to sell fish, most of them stay at Chimbai, Mahim or Khar Danda.

They are a very congenial lot, have fixed customers, extremely polite and gracious.

They are very fun loving people , but you have to drive a hard bargain, the best time to get fish is before they break for the day at about 1 oclock..

Than they give the fish to you at almost throw away price.

Sundays are really crowded.

On one side of the fish market a few women sell shell fish and live jumbo crabs.

The most in demand are Pomfrets, prawns , ravas and gol, bangdas or sardines , bombay ducks..

You hardly see lobsters here..

I have been coming to this market , which is now close to my house since 25 years.

A little ahead is the onoion sellers and beyond that the beef market .

Next to the beef market is the mutton market and the chicken shops.

Bandra Bazar is the most neglected Municipal market in Mumbai..filthy , dirty and totally unhygeinic during the rains when the entire market becomes inundated.

Broken Houses Broken Dreams

picture of pereira wadi bandra

tute hue gharon mein
yeh dilon ko kya basaienge
enka bas chale to paise ke liye
aap ki rooh ko bhi nigal jaienge
gharibi hatao siyasat ko bachao
tower aur shopping mall banao
kehke 15 august ko azadi
ka nara lagate hue
yeh apki kabar se guzar jayenge

searching for hope in a few coins in a beggars bowl

a leprous body
a leprous soul

The Leper Lady of Boran Road Wont See Big Boss

A show that makes me puke
a show for the sake of humanity
vociferously voyeuristic
they must certainly nuke

I recently quit my job.. now not on hire ..

i am very
good at
what i
so i cannot
be fired
i did not
want to
be a slave
to my bosses
whims fancies
i quit the job
i finally retired

Even Jesus In My Area Talks To Empty Chairs

to hon minister
with an angry
stare potholes
overflowing gutters
open garbage mounds
is that all you have
to share for the public
dont you have time
to spare ..why dosent
your party care ..
why are deleted dreams
on the soul of humanity
not repaired ..all kinds of
ghotala all kinds of gates
scams corruption ministerji
please dont swear
i am jesus christ these are
my people their pain
to me they have bared
you give them false promises
i give them hope fresh air

to my good friend randall der from montreal

Dried Bombay Ducks And My Cosmic Rings

On my hand ..on the thumb Lapis Lazuli Mueh Najaf Dure Najaf Mashad turqoise rare one .. I got it from a Irani seller for rs 500 and finally Nishapur turqoise ..

On my wrist turquoise from Jaipur cheap quality only for showbazi..


Leprosy by firoze shakir photographerno1
Leprosy, a photo by firoze shakir photographerno1 on Flickr.

Leprosy, or Hansen's disease, is a chronic infectious disease caused by the bacterium Mycobacterium leprae.[1] Leprosy is primarily a granulomatous disease of the peripheral nerves and mucosa of the upper respiratory tract; skin lesions are the primary external symptom.[2] Left untreated, leprosy can be progressive, causing permanent damage to the skin, nerves, limbs, and eyes. Contrary to popular conception,[3] leprosy does not cause body parts to simply fall off, and it differs from tzaraath, the malady described in the Hebrew scriptures and previously translated into English as leprosy.[4]

Historically, leprosy has affected humanity since at least 600 BC, and was well-recognized in the civilizations of ancient China, Egypt and India.[5] In 1995, the World Health Organization (WHO) estimated that between two and three million people were permanently disabled because of leprosy.[6] Although the forced quarantine or segregation of patients is unnecessary—and can be considered unethical—a few leper colonies still remain around the world, in countries such as India, Japan, Egypt, and Vietnam.

The age-old social stigma associated with the advanced form of leprosy lingers in many areas, and remains a major obstacle to self-reporting and early treatment. Effective treatment for leprosy appeared in the late 1930s with the introduction of dapsone and its derivatives. However, leprosy bacilli resistant to dapsone gradually evolved and became widespread, and it was not until the introduction of multidrug therapy (MDT) in the early 1980s that the disease could be diagnosed and treated successfully within the community

The clinical symptoms of leprosy vary but primarily affect the skin, nerves, and mucous membranes.[7] Patients with this chronic infectious disease are classified as having paucibacillary Hansen's disease (tuberculoid leprosy), multibacillary Hansen's disease (lepromatous leprosy), or borderline leprosy.

Contrary to popular belief, Hansen's bacillus does not cause rotting of the flesh; rather, a long investigation by Paul Brand yielded that insensitivity in the limbs extremities was the reason why unfelt wounds or lesions, however minute, lead to undetected deterioration of the tissues, the lack of pain not triggering an immediate response as in a fully functioning body.[8] Recently, leprosy has also emerged as a problem in HIV patients on antiretroviral drugs.[9]

Mycobacterium leprae is the causative agent of leprosy.[2] An intracellular, acid-fast bacterium, M. leprae is aerobic, gram-positive, and rod-shaped, and is surrounded by the waxy cell membrane coating characteristic of Mycobacterium species.[10]

Due to extensive loss of genes necessary for independent growth, M. leprae is unculturable in the laboratory, a factor which leads to difficulty in definitively identifying the organism under a strict interpretation of Koch's postulates.[11] The use of non-culture-based techniques such as molecular genetics has allowed for alternative establishment of causation.

The exact mechanism of transmission of leprosy is not known: prolonged close contact and transmission by nasal droplet have both been proposed, and, while the latter fits the pattern of disease, both remain unproven.[12] The only other animals besides humans known to contract leprosy are the armadillo, chimpanzee, sooty mangabey, and cynomolgus macaque.[13] The bacterium can also be grown in the laboratory by injection into the footpads of mice.[14] There is evidence that not all people who are infected with M. leprae develop leprosy, and genetic factors have long been thought to play a role, due to the observation of clustering of leprosy around certain families, and the failure to understand why certain individuals develop lepromatous leprosy while others develop other types of leprosy.[15] It is estimated that due to genetic factors, only 5 percent of the population is susceptible to leprosy.[16] This is mostly because the body is naturally immune to the bacteria, and those persons who do become infected are experiencing a severe allergic reaction to the disease. However, the role of genetic factors is not entirely clear in determining this clinical expression. In addition, malnutrition and prolonged exposure to infected persons may play a role in development of the overt disease.

The incubation period for the bacteria can last anywhere from two to ten years.

The most widely held belief is that the disease is transmitted by contact between infected persons and healthy persons.[17] In general, closeness of contact is related to the dose of infection, which in turn is related to the occurrence of disease. Of the various situations that promote close contact, contact within the household is the only one that is easily identified, although the actual incidence among contacts and the relative risk for them appear to vary considerably in different studies. In incidence studies, infection rates for contacts of lepromatous leprosy have varied from 6.2 per 1000 per year in Cebu, Philippines[18] to 55.8 per 1000 per year in a part of Southern India.[19]

Two exit routes of M. leprae from the human body often described are the skin and the nasal mucosa, although their relative importance is not clear. It is true that lepromatous cases show large numbers of organisms deep down in the dermis. However, whether they reach the skin surface in sufficient numbers is doubtful. Although there are reports of acid-fast bacilli being found in the desquamating epithelium of the skin, Weddell et al have reported that they could not find any acid-fast bacilli in the epidermis, even after examining a very large number of specimens from patients and contacts.[20] In a recent study, Job et al found fairly large numbers of M. leprae in the superficial keratin layer of the skin of lepromatous leprosy patients, suggesting that the organism could exit along with the sebaceous secretions.[21]

The importance of the nasal mucosa was recognized as early as 1898 by Schäffer, particularly that of the ulcerated mucosa. [22] The quantity of bacilli from nasal mucosal lesions in lepromatous leprosy was demonstrated by Shepard as large, with counts ranging from 10,000 to 10,000,000.[23] Pedley reported that the majority of lepromatous patients showed leprosy bacilli in their nasal secretions as collected through blowing the nose.[24] Davey and Rees indicated that nasal secretions from lepromatous patients could yield as much as 10 million viable organisms per day.[25]

The entry route of M. leprae into the human body is also not definitely known. The two seriously considered are the skin and the upper respiratory tract. While older research dealt with the skin route, recent research has increasingly favored the respiratory route. Rees and McDougall succeeded in the experimental transmission of leprosy through aerosols containing M. leprae in immune-suppressed mice, suggesting a similar possibility in humans.[26] Successful results have also been reported on experiments with nude mice when M. leprae were introduced into the nasal cavity by topical application. [27] In summary, entry through the respiratory route appears the most probable route, although other routes, particularly broken skin, cannot be ruled out. The CDC notes the following assertion about the transmission of the disease: "Although the mode of transmission of Hansen's disease remains uncertain, most investigators think that M. leprae is usually spread from person to person in respiratory droplets."[28]

In leprosy both the reference points for measuring the incubation period and the times of infection and onset of disease are difficult to define; the former because of the lack of adequate immunological tools and the latter because of the disease's slow onset. Even so, several investigators have attempted to measure the incubation period for leprosy. The minimum incubation period reported is as short as a few weeks and this is based on the very occasional occurrence of leprosy among young infants. [29] The maximum incubation period reported is as long as 30 years, or over, as observed among war veterans known to have been exposed for short periods in endemic areas but otherwise living in non-endemic areas. It is generally agreed that the average incubation period is between 3 to 5 years.

[edit] Treatment

MDT patient packs and blistersUntil the development of dapsone, rifampicin, and clofazimine in the 1940s, there was no effective cure for leprosy. However, dapsone is only weakly bactericidal against M. leprae and it was considered necessary for patients to take the drug indefinitely. Moreover, when dapsone was used alone, the M. leprae population quickly evolved antibiotic resistance; by the 1960s, the world's only known anti-leprosy drug became virtually useless.

The search for more effective anti-leprosy drugs than dapsone led to the use of clofazimine and rifampicin in the 1960s and 1970s.[30] Later, Indian scientist Shantaram Yawalkar and his colleagues formulated a combined therapy using rifampicin and dapsone, intended to mitigate bacterial resistance.[31] Multidrug therapy (MDT) and combining all three drugs was first recommended by a WHO Expert Committee in 1981. These three anti-leprosy drugs are still used in the standard MDT regimens. None of them is used alone because of the risk of developing resistance.

Because this treatment is quite expensive, it was not quickly adopted in most endemic countries. In 1985 leprosy was still considered a public health problem in 122 countries. The 44th World Health Assembly (WHA), held in Geneva in 1991 passed a resolution to eliminate leprosy as a public health problem by the year 2000 — defined as reducing the global prevalence of the disease to less than 1 case per 100,000. At the Assembly, the World Health Organization (WHO) was given the mandate to develop an elimination strategy by its member states, based on increasing the geographical coverage of MDT and patients’ accessibility to the treatment.

The WHO Study Group's report on the Chemotherapy of Leprosy in 1993 recommended two types of standard MDT regimen be adopted.[32] The first was a 24-month treatment for multibacillary (MB or lepromatous) cases using rifampicin, clofazimine, and dapsone. The second was a six-month treatment for paucibacillary (PB or tuberculoid) cases, using rifampicin and dapsone. At the First International Conference on the Elimination of Leprosy as a Public Health Problem, held in Hanoi the next year, the global strategy was endorsed and funds provided to WHO for the procurement and supply of MDT to all endemic countries.

MDT anti-leprosy drugs: standard regimensBetween 1995 and 1999, WHO, with the aid of the Nippon Foundation (Chairman Yōhei Sasakawa, World Health Organization Goodwill Ambassador for Leprosy Elimination), supplied all endemic countries with free MDT in blister packs, channelled through Ministries of Health. This free provision was extended in 2000 with a donation by the MDT manufacturer Novartis, which will run until at least the end of 2010. At the national level, non-government organizations (NGOs) affiliated to the national programme will continue to be provided with an appropriate free supply of this WHO supplied MDT by the government.

MDT remains highly effective and patients are no longer infectious after the first monthly dose.[5] It is safe and easy to use under field conditions due to its presentation in calendar blister packs.[5] Relapse rates remain low, and there is no known resistance to the combined drugs.[5] The Seventh WHO Expert Committee on Leprosy, [33] reporting in 1997, concluded that the MB duration of treatment—then standing at 24 months—could safely be shortened to 12 months "without significantly compromising its efficacy."

Persistent obstacles to the elimination of the disease include improving detection, educating patients and the population about its cause, and fighting social taboos about a disease for which patients have historically been considered "unclean" or "cursed by God" as outcasts. Where taboos are strong, patients may be forced to hide their condition (and avoid seeking treatment) to avoid discrimination. The lack of awareness about Hansen's disease can lead people to falsely believe that the disease is highly contagious and incurable.

The ALERT hospital and research facility in Ethiopia provides training to medical personnel from around the world in the treatment of leprosy, as well as treating many local patients. Surgical techniques, such as for the restoration of control of movement of thumbs, have been developed there.

[edit] Prevention
A single dose of rifampicin is able to reduce the rate of leprosy in contacts by 57% to 75%.[34][35]

BCG is able to offer a variable amount of protection against leprosy as well as against tuberculosis.[36][37]

[edit] Epidemiology

World distribution of leprosy, 2003.Worldwide, two to three million people are estimated to be permanently disabled because of Hansen's disease.[6] India has the greatest number of cases, with Brazil second and Myanmar third.

In 1999, the world incidence of Hansen's disease was estimated to be 640,000; in 2000, 738,284 cases were identified. In 1999, 108 cases occurred in the United States. In 2000, the World Health Organization (WHO) listed 91 countries in which Hansen's disease is endemic. India, Myanmar and Nepal contained 70% of cases. In 2002, 763,917 new cases were detected worldwide, and in that year the WHO listed Brazil, Madagascar, Mozambique, Tanzania and Nepal as having 90% of Hansen's disease cases.

According to recent figures from the WHO, new cases detected worldwide have decreased by approximately 107,000 cases (or 21%) from 2003 to 2004. This decreasing trend has been consistent for the past three years. In addition, the global registered prevalence of HD was 286,063 cases; 407,791 new cases were detected during 2004.

Hansen's disease is tracked by the Centers for Disease Control and Prevention (CDC). Its prevalence in the United States is believed to be rising and underreported.[38] Although the number of cases worldwide continues to fall, pockets of high prevalence continue in certain areas such as Brazil, South Asia (India, Nepal), some parts of Africa (Tanzania, Madagascar, Mozambique) and the western Pacific.

[edit] Risk groups
At highest risk are those living in endemic areas with poor conditions such as inadequate bedding, contaminated water and insufficient diet, or other diseases (such as HIV) that compromise immune function. Recent research suggests that there is a defect in cell-mediated immunity that causes susceptibility to the disease. Less than ten percent of the world's population is actually capable of acquiring the disease[citation needed]. The region of DNA responsible for this variability is also involved in Parkinson's disease, giving rise to current speculation that the two disorders may be linked in some way at the biochemical level. In addition, men are twice as likely to contract leprosy as women.

[edit] Disease burden
Although annual incidence—the number of new leprosy cases occurring each year—is important as a measure of transmission, it is difficult to measure in leprosy due to its long incubation period, delays in diagnosis after onset of the disease and the lack of laboratory tools to detect leprosy in its very early stages.

Instead, the registered prevalence is used. Registered prevalence is a useful proxy indicator of the disease burden as it reflects the number of active leprosy cases diagnosed with the disease and retrieving treatment with MDT at a given point in time. The prevalence rate is defined as the number of cases registered for MDT treatment among the population in which the cases have occurred, again at a given point in time.[39]

New case detection is another indicator of the disease that is usually reported by countries on an annual basis. It includes cases diagnosed with onset of disease in the year in question (true incidence) and a large proportion of cases with onset in previous years (termed a backlog prevalence of undetected cases). The new case detection rate (NCDR) is defined by the number of newly detected cases, previously untreated, during a year divided by the population in which the cases have occurred.

Endemic countries also report the number of new cases with established disabilities at the time of detection, as an indicator of the backlog prevalence. However, determination of the time of onset of the disease is generally unreliable, is very labour-intensive and is seldom done in recording these

There is some confusion over classification because the WHO replaced an older, more complicated classification system with a simpler system that identifies two subtypes of leprosy - paucibacillary and multibacillary. The older system included six categories: Indeterminate Leprosy, Borderline Tuberculoid Leprosy, Midborderline Leprosy, Borderline Lepromatous Leprosy, Lepromatous Leprosy, and Tuberculoid Leprosy.

Paucibacillary leprosy encompasses indeterminate, tuberculoid, and borderline tuberculoid leprosy. It is characterized by one or more hypopigmented skin macules and anaesthetic patches, i.e., damaged peripheral nerves that have been attacked by the human host's immune cells.

Multibacillary leprosy includes midborderline, borderline lepromatous, and lepromatous leprosy. It is associated with symmetric skin lesions, nodules, plaques, thickened dermis, and frequent involvement of the nasal mucosa resulting in nasal congestion and epistaxis (nose bleeds) but typically detectable nerve damage is late.

Borderline leprosy is of intermediate severity and is the most common form. Skin lesions resemble tuberculoid leprosy but are more numerous and irregular; large patches may affect a whole limb, and peripheral nerve involvement with weakness and loss of sensation is common. This type is unstable and may become more like lepromatous leprosy or may undergo a reversal reaction, becoming more like the tuberculoid form.

Promises to Keep

the august
leaders at the back
tell him live in mumbai
dont be afraid
let the party
come to power
next elections
we will give you
a timely upgrade
you may be from
mayawati or laluland
but you are desi made
ignore the political tirade
register with the municipality
see that your dues are paid
illegal hawking
should no be custom made
without bhaiyyas
up the chimney goes
our commercial trade

Grieving Hands

a single
a pain expands
the poor the needy
the neglected
of our mother land

Bandra Bezar Road

choked up gutters
filth more filth
the destiny
of bandra bazar road
the coming elections
the netas will come
with hands
as of old
a misery of
five years
in silent

ek naya kamar tod

Searching For The Unmarked Grave of a Beggar Poet at Banda Bazar Road

Agar Camera Nahi Ata Kinara Mil Gaya Hota..

I share other peoples dreams through the camera in the bargain i lost my a wold of illusions all alone ..god gave her beauty intellect alas he gave her a heart of stone.. i shoot others more fortunate than me a pain i have borne ...i became one of them.. a dream adorn...she will never be mine she has sworn....leaving my nest to a gilded cage she has flown..

Le Bijou - Narayan and Me Bandra Hill Road

Mithi River of Mumbai

from wikipedia

The Mithi River (aka Mahim River) is a river in Salsette Island, the island of the city of Mumbai. It is a confluence of tail water discharges of Powai and Vihar lakes. The river is seasonal and rises during the monsoons. The overflowing lakes also contribute to the river flow which is stopped by a dam in other times. During this season the river is a favourite with the anglers who catch large fish that have escaped from the lakes. Fishing is banned there.[citation needed]
The river is also a natural drainage channel which carries the excess waters during the monsoons. However it is filthy mainly due to the presence of slums and the discharge of industrial effluents.[citation

The river originates from the overflow of Vihar Lake and also receives the overflows from the Powai Lake about 2 km later. It flows for a total of 15 km before it meets the Arabian Sea at Mahim Creek flowing through residential and industrial complexes of Powai, Saki Naka, Kurla, Kalina, Vakola, Bandra-Kurla complex, Dharavi and Mahim. The river has an average width of 5 m in the upper reaches, has been widened to 25 m in the middle reaches and up to 70 m in the lower reaches after the 26 July 2005 deluge (944 mm in 24 h on 26 July 2005).[citation needed]

It is also less well known that the Mahim bay area, where Mithi River meets Arabian Sea is a nominated bird sanctuary where migratory birds come for nesting. This part is full of mangroves. When the river was not as polluted as it is today, it used to serve as an important storm water drain for Mumbai but as it has been used as a sewer over the years, its importance as a storm water drain has reduced and on the contrary, it poses as a hazard during high tide bringing polluted water into the city.
[edit]Environmental degradation

Very often, and this continues to happen, citizens dump raw sewage, industrial waste and municipal waste into the river, unchecked. Besides this, illegal activities like washing vessels, animals and oily drums, discharge of unauthorised hazardous waste are also carried out along the course of this river. Cattle sheds in some areas contribute animal waste. Barrel cleaners, scrap dealers and others dump sludge oil, effluent and garbage in the river. The organic waste, sludge and garbage dumping has reduced the carrying capacity of the river. The water with mixture of sewage and industrial waste is a threat to marine life. The river bed is full of sludge, garbage and vegetation growth like water hyacinth in many parts.

In 2006, the Supreme court of India passed an order directing the state government to remove encroachments on all water bodies.[citation needed]
The Municipal Corporation of Greater Mumbai has undertaken a cleanliness drive lately so that the floods of July 26, 2005 are not repeated.
An environmental group has been formed by Rajendra Singh, an award winning conservationist in 2009.[1] It aims to revive the dying river.

Many young entrepreneurs in and around Mumbai are now aggressively involved with cause of Mithi River, and creating awareness on a global scale as the government of India has once again started ignoring this extremely important issue.[2] In 2009, environmentalist and Magsaysay Award winner, Rajendra Singh lead a yatra, of a group of environmentalist and NGOs, through Mumbai city along the endangered Mithi river to highlight its problems.[3]
Recently a Contemporary Art show was held to create major awareness about dire situation of Mithi River in Bombay by "Chintan Upadhyay" titled Khatti - Mithi[4]

The Recycling Bin

they have
used you
they push
you in
a kick
on your ass
a punch
on your chin
here they
only worship
those who win
losers ..gone
for a toss
gone for a spin

Bollywoods Most Wanted Positive Spam..

Marziya Lap Top Kid

Bandra Bazar Road - A sacrificial goat

So many MP's , MLA's
have come and gone
so many will soon come
will be gone
tiling re-opening
gutters from dusk to dawn
a vote bank of
eunuch residents
including myself
as we mourn
neglected bandra bazar
a sacrificial goat
a political pawn
everybody shitting
on a manicured lawn
a virgin rose
by a circumstantial thorn
a dying heritage
the queen of suburbs
to the newly born
party politics
Congress I
Shiv Sena

promises made
hopes outworn
says the
reluctant foetus
I'd rather
not be born
in bazar road
garbage porn

Hot Cuppa Chai

bid your bad dreams
unpaid rent of your
paying guest
good bye
meet your land ladys
daughter on the sly
wipe the tear from her eye
impregnate her fast
the house of rent
will be your
permanent address by and by
to beat the sky rocketing
prices of property in mumbai
the brain works faster
with a cutting
or a hot cuppa chai
a thought
the slimy
real estate broker
wont deny

The Bhaiyya and the Badasaab

The Wheel of Distrust

man for
a womans
flesh does lust
his dreams
within her
at its worst
ashes to ashes
dust to dust
i rust
my sincerity
my honesty
my faith
she does
not trust
my love
last and first

The Mango Man from UP

his land
his family
he is here
in mumbai
his dreams
his destiny
they call him a bhaiyya
this mango man from UP
he too is a mumbaikar
bearing the political
heat and humdity
selling appus
from ratnagiri

akhilesh bhaiyya
yeh kaisi majboori
bchte hain am
aur gale par hai churi

I Am Updating My Street Photography Set At 35618 images

241,405 items / 2,027,691 views

As I shoot and add my pictures to respective sets at Flickr I have over 558 sets , some pictures not shot on the streets ended up on this set that I am correcting as I have time on hand.

And to think of it all my religion pictures of various faith processions are street pictures that but I did not add them here it would be tough managing them.

Most of my street pictures belong to Bandra Bazar Road and once the CRZ opens up Bandra Bazar will be the next Manhattan.. these are my words of prophecy as a DAm Madar Malang ..

Rates will skyrocket and whatever Mr Pritish Nandy whom I follow on Twitter feels about property rates coming down this will never happen as long as Congress remains in powere here or at the center.. the remote control lies in the hands of the builders heck what am I saying most of the netas are fronts for builders or vice versa.

But this is not a political blog a poor beggar poets blog..

It is connected by arteries veins of Twitter and Facebook.. some cerebral fluid reaches pinterest too..

I Became a Street Photographer ..

241,405 items / 2,027,659 views

Because pictures like these are shunned by fine art arrogant pompous photographers who call the shots at dead defunct camera clubs .. living million light years away farting their old time fucked views on the almost dying soul of photography.

One camera club salon pictures in the the newsletter is almost similar in resemblance to a newsletter in JhumriTalaya.

For once though I am a product of a camera club in Mumbai I wont ever let my grand children ever become part of that culture that does not expect change does not like change and always looked down on bloggers we dont sell our souls we give it free as charity on the internet.

Now most of them are finally understanding the power of the Internet and coming out of their blinkered closet .. and I say this in good faith no personal animosity.. camera clubs some of them have become totally despotic and political.. why the clubs were created has been forgotten.. now it is about big bucks .. this is my personal opinion and you or the guy next to you can never change it.

My relationship with camera club died the day my Guru Prof BW Jatkar died.RIP Om Shanti Om

May his Soul and the Soul of All Camera Clubs rest in peace .

My Grand Daughters ,, I taught them the one cardinal rule of my personal street photography.. they being brilliant dont need translation,


Beyond My Reach

a pebble
on the sands
the waves
of life
a mortal
the lord
i beseech
in a classroom
i teach
but my
lies across
the ocean
million miles
my reach
as a poet
to her
woman's soul
like leech

An Ode To Empty Chairs

241,405 items / 2,027,615 views

of presidents
are not spared
dirty harry
gives them
the scare
a thought
i share
the good
the bad
the ugly
a poetic
i bare
all in all
house people
come and go
it is his country
his people
for whom he cares
clint eastwood
in the line of fire
breathing words
rumbustious air

to Anthony Poesy
Brother Poet

Media Thinks Bloggers Dont Shoot Pictures ,, No We Dont Shoot Crap as Pictures

Lalbagh Cha Raja 75 Glorious Years

Sunil Joshi My Dear Friend

I Shoot The Humility of Lalbagh Chya Raja..